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Rethinking What Matters

Across fields, the same question leads researchers in new directions.

  • Research
  • JooHyeon Heo
  • 2026.09.01
  • 5508

Rethinking What Matters

《Editor's Note: What does it take to make something work better? The answer may be to pare back what is unnecessary, rethink how existing parts are arranged, find another route around a constraint, or widen the frame of the problem itself. This month's UNIST research offers answers in many forms—from leaner AI systems and redesigned material structures to an unexpected route for electron transfer and a broader view of cancer drug resistance. Some of those answers run counter to what we might expect.   




Less Can Be Enough  


“How much does AI really need?”  


AI has spent years scaling up. But recent UNIST research asks a different questionhow much can be taken away while keeping what matters?


At ACL 2026, researchers introduced GMoE, an architecture that shares experts across layers rather than repeatedly storing similar ones. It cut model parameters by about 63 percent, from 549 million to 204 million, while average accuracy remained virtually unchanged—39.51 percent, compared with 39.55 percent for the baseline.


The next target was data. Presented at ICML 2026, CSOR removes redundant training images while preserving meaningful variation within each identity. Using roughly half the original dataset, models retained more than 95 percent of full-data performance in re-identification (ReID) tasks.


A third study moved to hardware. An automated design framework for logic-in-memory (LiM) computing optimized the placement and routing of computing elements, cutting computation cycles by 19.6 percent under area constraints.


            GMoE—Expert Sharing for Smaller AI Models (ACL 2026)

■             CSOR—Less Training Data for ReID AI (ICML 2026  )

■              LiM—Smarter Chip Design (IEEE TCAD)




Arrangement Matters


“In materials, where can matter as much as what.”


More iron, less corrosion. Iron (Fe) is one of magnesium's most damaging impurities, yet UNIST researchers produced a Mg alloy containing more than six times as much Fe as ultra-high-purity Mg—and it still corroded more slowly. Instead of removing the impurity, the team added scandium (Sc) to form core–shell particles that isolate Fe-rich regions from the surrounding Mg. The corrosion rate fell from 22.2 to 0.38 millimeters per year, nearly a 60-fold reduction.


At the molecular scale, thermal annealing changed the packing of an organic semiconductor, more than tripling its absorption dissymmetry and even reversing its preferred handedness of circularly polarized light (CPL). The resulting near-infrared (NIR) photodetector achieved a dissymmetry factor of 0.1 at 850 nanometers.


The scale shifts again in perovskite solar cells (PSCs). The same high-efficiency formulation crystallized differently depending on the buried interface beneath it. Tailoring the chloride chemistry to the inverted architecture produced a device with 26.3 percent efficiency and a fill factor (FF) of up to 86.8 percent.


■                Mg Alloy—Shielding Fe to Slow Corrosion (Science Advances) 

■                 NIR Photodetector—Turning Molecular Packing (Advanced Science)

■                 PSCs—Why Architecture Changes Performance (Joule)




Another Way Through


“How spin opens a new route for electron transfer.”


Conventional energetics suggested that electron transfer should be unfavorable. Yet in manganese (Mn)-doped quantum dots (QDs), spin-exchange interactions enabled transfer more than ten times faster than in undoped QDs. The mechanism could help capture energetic hot electrons before their excess energy dissipates—a longstanding challenge in photocatalysis and solar-energy conversion.


■               Hot Electrons—Spin-Enabled Transfer (Nature Communications) 




Beyond the Tumor


“The cells helping cancer resist treatment.”


Some of the cells helping breast cancer resist treatment are not part of the tumor at all. UNIST researchers found that nearby fat cells can become cancer-associated adipocytes, which release complement factor D and activate survival signaling in cancer cells. 


In mouse models, disrupting this TRAP1-driven communication improved the response to chemotherapy, pointing to surrounding adipocytes as potential therapeutic targets alongside the tumor itself.


■                 Breast Cancer—How Nearby Fat Cells Drive Drug Resistance (STTT) 





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